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Q1. Target site duplications are seen at the sites of transposition of bacterial transposes and both LTR and non-LTR type eukaryotic retro transposes. How do these duplications arise and what do they propose in terms of mechanisms of transposition?

Q2. Could you help to understand FEV1/FVC and how the 'expected percentage' of this figure relates to respiratory problems?

Also with reference to the 3 zones of the lungs, zone 3 is more highly per fused because of bigger area and gravity but which zone has more ventilation and why explains?

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