Question: Both proto-oncogenes and tumor suppressor genes can interact with transcription factors involved cell division. What is the fundamental difference between how the two function? Think ‘end result'.
Question: For Exam 1, we defined retinoblastoma as an autosomal dominant characteristic. We know that it is caused by a loss of function mutation for Rb. The Rb gene is autosomal recessive. How can you reconcile those two (seemingly) different definitions?